primary antibodies against insulin (Proteintech)
96
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Proteintech
primary antibodies against insulin
Primary Antibodies Against Insulin, supplied by Proteintech, used in various techniques. Bioz Stars score: 96/100, based on 330 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/primary+antibodies+against+insulin/FOXO1+Antibody/pm41101618-78-11-35
Average 96 stars, based on 330 article reviews
Primary Antibodies Against Insulin, supplied by Proteintech, used in various techniques. Bioz Stars score: 96/100, based on 330 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/primary+antibodies+against+insulin/FOXO1+Antibody/pm41101618-78-11-35
Average 96 stars, based on 330 article reviews
primary antibodies against insulin - by Bioz Stars,
2026-10
96/100 stars
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Blocking Assay:Article Title: Di-(2-ethylhexyl) phthalate exposure disrupts glucose homeostasis through inhibition of PI3K/Akt/FoxO1 pathway triggering β-cell dedifferentiation in male rats. Article Snippet: Di-(2-ethylhexyl) phthalate (DEHP), a widely used environmental endocrine disruptor, has been associated with an increased risk of Type 2 Diabetes Mellitus (T2DM).. However, it remains unclear whether exposure to DEHP and its active metabolite can disrupt metabolic homeostasis by inducing pancreatic β-cell dedifferentiation.. To evaluate the effects of DEHP on glucose homeostasis and β-cell dedifferentiation, we established an in vivo exposure model using male SD rats, administered with varying concentrations of DEHP (0, 2, 20 and 200 mg/kg/day) over six weeks. Article Title: The effect of aspartame on accelerating caspase-dependent apoptosis of pancreatic islet via ZIPK/STAT3/caspase 3 signaling pathway. Article Snippet: Aspartame (ASP) as an important sugar substitute is widely used in pharmaceutical and food processing.. Here, we compared the effects of ASP and sucrose on mice pancreatic islet cells in vivo and observed that ASP with the condition of high concentration and long-term exposure (HASP) could cause insulin secretion (500 mg/kg for 1 month).. Next, we conducted iTRAQ mass spectrometry to profile the global phosphoproteome and found that phosphorylation of zipper-interacting protein kinase (ZIPK) in murine pancreatic islet tissues were induced at Thr197, Thr242, Thr282, and Ser328 by high-sucrose (HS) treatment, but only induced at Thr197 and Ser328 by HASP treatment. Incubation:Article Title: Di-(2-ethylhexyl) phthalate exposure disrupts glucose homeostasis through inhibition of PI3K/Akt/FoxO1 pathway triggering β-cell dedifferentiation in male rats. Article Snippet: Di-(2-ethylhexyl) phthalate (DEHP), a widely used environmental endocrine disruptor, has been associated with an increased risk of Type 2 Diabetes Mellitus (T2DM).. However, it remains unclear whether exposure to DEHP and its active metabolite can disrupt metabolic homeostasis by inducing pancreatic β-cell dedifferentiation.. To evaluate the effects of DEHP on glucose homeostasis and β-cell dedifferentiation, we established an in vivo exposure model using male SD rats, administered with varying concentrations of DEHP (0, 2, 20 and 200 mg/kg/day) over six weeks. |